Cutaneous Dysbiosis and Inflammatory Skin Diseases
- In:
Clinical Practice
Cutaneous Dysbiosis and Inflammatory Skin Diseases presented by Adam Friedman MD
- Certain ribotypes of C. acnes convert sebum to short-chain fatty acids, which initiate the inflammatory acne cascade via activation of toll-like receptors.
- Narrow spectrum antibiotics in acne treatment reduce the risk of altering both the gut and skin microbiome.
- Application of a prebiotic moisturizer results in expansion of cutaneous microbial diversity, which results in decreased staph colonization on the skin, which may prove beneficial in atopic dermatitis and acne patients.